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Home » Electroconvulsive Therapy

Psychiatric Times. Vol. 29 No. 8
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PRACTICE PERSPECTIVES 

The Occupation of Psychiatry?

by William M. Glazer, MD | July 31, 2012
Dr Glazer is President of Glazer Medical Solutions in Chilmark, Mass. He receives funding from Eli Lilly and Merck.

Whitaker refers to the research of Harrow and Jobe9 in his contention that long-term antipsychotic treatment causes increased pathology. The Harrow and Jobe study is a marvelous 15-year follow-up of 64 schizophrenic patients—not an easy study to perform, in fact, a true labor of love. Twenty of these patients did not receive antipsychotic medications, and the researchers noted that their outcomes were more likely to be better than what was seen in the 44 patients who received antipsychotics.

Is it the disease or the treatment?

Whitaker’s conclusion is that long-term use of antipsychotics is not necessary for or may be harmful to patients’ functioning. He dismisses the fact (brought out clearly by the study authors) that the untreated patients in this nonrandomized observational study were likely to have been self-selected (ie, they were healthier and elected not to take antipsychotics). Whitaker reverses cause and effect to sustain his argument. Standard clinical logic would conclude that the poor outcomes in the antipsychotic-treated patients were a function of their illness—not the treatment. (Interested readers may want to see E. Fuller Torrey’s excellent analysis of Whitaker’s “tortured logic” in the schizophrenia studies.10)

The Harrow and Jobe study, by virtue of its design, is severely limited in its ability to generate clear and conclusive evidence that antipsychotics cause or increase morbidity over time. Ask Harrow and Jobe if you don’t believe me. The value of the Harrow and Jobe study is at the level of hypothesis generation. It suggests that not all schizophrenia patients require long-term medications. The question is, which ones?

The same type of discussion can be applied to Whitaker’s treatment of antidepressants, antianxiety agents, and mood stabilizers. He consistently employs a reductionist mentality to reach the “astonishing and startling” (Amazon.com’s advertisement words for the book) conclusion that our drugs have caused an “epidemic” of increased psychopathology in the major psychiatric disorders.

What to make of all this?

Should we accept the analysis of a journalist who (1) to my knowledge, has not treated a patient or implemented a study and (2) reaches conclusions that run counter to well-established practice guidelines? Whitaker’s ideological viewpoint, which is implied throughout the book, is that our guidelines are inaccurate and driven by industry and our own need for income—that we are dishonest brokers. Beauty is in the eye of the beholder.

I would predict that if the authors of the studies on which Whitaker relied convened a meeting to critically discuss the book, the vast majority would vote against this journalist’s idiosyncratic conclusion.

No one will argue that there is a dearth of long-term studies, defined as longer than 2 years, of the enduring effects of psychiatric medications. If Whitaker’s work stimulates more long-term research, then it serves the field. But while we wait for that research, should we change our practice to fit this journalist’s thesis that our medications cause poor outcomes? My answer is no.

Occupying the doctor-patient relationship

As practitioners, we work with our patients and provide for them the best treatment that we can, based on their preference, available evidence, and our judgment. We work collaboratively with each patient to weigh the risk to benefit ratio of long-term prescriptions over time. Risk to benefit calculations are no easy task. It is difficult to identify whether one patient requires long-term treatment following a good response to short-term treatment. Ideally, these decisions proceed within a solid alliance that we establish with the patient and others. Successful alliances require trust.

Books (and blogs) that go out for public consumption alleging conspiracy among doctors that results in epidemics and offering a prejudiced review of select literature run the risk of distorting that trust. (I am interested in hearing about direct experiences that you may have had in which articles, blogs, or books of the kind that I have discussed have altered existing treatment plans—positively or negatively. Contact me at wglazer@glazmedsol.com or 305-293-3555.)

We and our patients do not need to have that trust hijacked by misguided, “astonishing and startling” conclusions presented in publications such as Anatomy of an Epidemic. It is our obligation to protect that trust.

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by Ronald Pies | July 31, 2012 9:22 PM EDT

Many thanks to Dr. Glazer for another debunking of Whitaker's thesis (Dr. Torrey's piece is also well worth reading). Whitaker's popularity is symptomatic of the current rush to judgment about psychiatric treatments, largely propelled by people with no scientific training, medical experience, or understanding of the numerous variables involved in outcome studies. (A similar theme emerges in the sensationalized lay publications on "the adaptive value"of major depressive disorder, and the supposed lack of efficacy of antidepressants).

This is not to say that we should ignore everything Whitaker, or other critics of psychiatry, have to tell us. As Dr. Torrey notes, some elements of Whitaker's critique (e.g., regarding over-use and over-prescription of some psychotropics, in some settings) need to be taken seriously. But, as I argue in my article, "Is there really an epidemic of psychiatric illness in the U.S.?" [Psychiatric Times] the notion that there is a growing "epidemic" of mental illness in this country is really an elaborate fiction.

Best regards, and many thanks, Bill!

Ron





References

1. Glazer WM. Rebuttal: questioning the validity of Anatomy of an Epidemic (Part I). Behavioral Healthcare. October 31, 2011. http://www.behavioral.net/article/rebuttal-questioning-validity-anatomy-epidemic. Accessed June 28, 2012.

2. Glazer WM. Rebuttal: questioning the validity of Anatomy of an Epidemic (Part II). Behavioral Healthcare. November 15, 2011. http://www.behavioral.net/article/rebuttal-questioning-validity-anatomy-epidemic-part-ii. Accessed June 29, 2012.

3. Angell M. The epidemic of mental illness and the illusions of psychiatry (2-part review). New York Review of Books. 2011.

4. Wilkinson RG, Pickett K. The Spirit Level: Why More Equal Societies Almost Always Do Better. London: Allen Lane; 2009.

5. Kessler RC, Demler O, Frank RG, et al. Prevalence and treatment of mental disorders, 1990 to 2003. N Engl J Med. 2005;352:2515-2523.

6. Substance Abuse and Mental Health Services Administration. Mental Health, United States, 2010. Rockville, MD: Substance Abuse and Mental Health Services Administration, US Dept of Health and Human Services; 2012. HHS publication (SMA) 12-4681.

7. Fava GA. Do antidepressant and antianxiety drugs increase chronicity in affective disorders? Psychother Psychosom. 1994;61:125-131.

8. El-Mallakh RS, Gao Y, Roberts JR. Tardive dysphoria: the role of long term antidepressant use in inducing chronic depression. Med Hypotheses. 2011;76:769-773.

9. Harrow M, Jobe TH. Factors involved in outcome and recovery in schizophrenia patients not on antipsychotic medications: a 15-year multifollow-up study. J Nerv Ment Dis. 2007;195:406-414.

10. Torrey EF. Anatomy of a nonepidemic—a review by Dr Torrey: How Robert Whitaker got it wrong. Treatment Advocacy Center. 2011. http://www.treatmentadvocacycenter.org/index.php?option=com_content&task=view&id=2085. Accessed June 28, 2012.


 
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